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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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DTSTART;VALUE=DATE:20240610
DTEND;VALUE=DATE:20240612
DTSTAMP:20260422T072537
CREATED:20240205T092642Z
LAST-MODIFIED:20240611T151556Z
UID:167545-1717977600-1718150399@www.bordeaux-neurocampus.fr
SUMMARY:NeuroPain Summer School - Open access lectures
DESCRIPTION:Venue: CARF (ex CGFB) \n\nThe « Neural circuits of pain: from physiology to pathology » summer school is open to doctoral students and post-doctoral fellows with an interest in neurology\, neuroscience\, molecular biology and electrophysiology. \nThis five-day programme will allow students to explore the physiological and pathological aspects of pain\, employing cutting-edge technologies. Renowned scientists will train participants for advancements in pain treatment. Course content includes advanced courses\, workshops on techniques as well as hands-on training. \nMonday June 10th \n\n8h30- Welcoming\, opening session. \n9h-9h50. Prof Rohini Kuner – « Neuronal ensembles underlying pain perception\, chronicity and interactions with negative emotions » \n9h55-10h45. Prof Greg Scherrer – « Neural circuits and therapeutics for pain unpleasantness and its cognitive modulation » \nCoffee Break \n11h-11H50. Prof Jing Wang  – « Applying a closed-loop brain-computer interface to study and treat pain » \n11h55-12h45. Prof Michelle Roche – « Understanding the crosstalk between Stress\, Mood and Pain » \nTuesday June 11th \n\n8h30-9h20 : Prof Tomoko Ohyama – « Nociception in Drosophila larvae. » \n9h25-10h15: Prof Simon Beggs – « The influence of pain in early life pain on adult pain circuits and behaviour. » \nCoffee Break \n10h30-11h20: Prof Steven Prescott – « Importance of sensory neuron excitability for neural coding » \nMore details\nhttps://bss-neuropain.u-bordeaux.fr/en/ \nContact\nbf-bss.neuropain@u-bordeaux.fr \n
URL:https://www.bordeaux-neurocampus.fr/event/neuropain-lectures/
CATEGORIES:Pour les scientifiques
ATTACH;FMTTYPE=image/png:https://www.bordeaux-neurocampus.fr/wp-content/uploads/2024/02/summer-school-pain-e1711558666214.png
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DTSTART;TZID=Europe/Paris:20240610T140000
DTEND;TZID=Europe/Paris:20240610T140000
DTSTAMP:20260422T072537
CREATED:20240502T131540Z
LAST-MODIFIED:20240523T123729Z
UID:171285-1718028000-1718028000@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - George Koob
DESCRIPTION:Venue: Centre Broca \n\nGeorge F. Koob\, Ph. D.\nDirector\, National Institute on Alcohol Abuse and Alcoholism\nSenior Investigator\, National Institute on Drug Abuse\nBethesda\, MD \nInvited by Serge Ahmed (INCIA) \nTitle\nNeurobiology of Alcohol and Opioid Addiction: A hyperkatifeia\, negative reinforcement\, and allostasis perspective  \nAbstract\nAddiction is a chronically relapsing disorder characterized by compulsive drug seeking that is hypothesized to derive from multiple sources of motivational dysregulation\, one of which is negative reinforcement driven by the emotional pain of alcohol withdrawal and protracted abstinence. The construct of negative reinforcement is defined as drug taking that alleviates a negative emotional state or hyperkatifeia (pain\, hypohedonia\, dysphoria\, anxiety\, hyperalgesia\, irritability\, and sleep disturbances) that is created by drug abstinence following excessive drug consumption. In humans\, and animal models\, repeated misuse of drugs results in hyperkatifeia as reflected in increased reward thresholds\, decreased pain thresholds\, anxiety-like and dysphoric-like responses. Such hyperkatifeia that drives negative reinforcement is hypothesized to derive from dysregulation of key neurochemical circuits within the brain reward and stress systems. Thus\, compelling evidence exists to argue that hyperkatifeia triggered by acute excessive drug intake\, is sensitized during the development of compulsive alcohol taking with repeated withdrawal\, persists into protracted withdrawal\, and contributes to the development and persistence of compulsive alcohol seeking. The construct of negative reinforcement fits a framework of allostasis where other allostatic loads such as genetics/ epigenetics\, childhood trauma\, and social determinants of health join hyperkatifeia in driving addiction. \n  \n  \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-george-koob/
CATEGORIES:A la une,Pour les scientifiques,Séminaire Impromptu
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