BEGIN:VCALENDAR
VERSION:2.0
PRODID:-//Bordeaux Neurocampus - ECPv4.9.10//NONSGML v1.0//EN
CALSCALE:GREGORIAN
METHOD:PUBLISH
X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
BEGIN:VTIMEZONE
TZID:Europe/Paris
BEGIN:DAYLIGHT
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
TZNAME:CEST
DTSTART:20260329T010000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
TZNAME:CET
DTSTART:20261025T010000
END:STANDARD
END:VTIMEZONE
BEGIN:VEVENT
DTSTART;VALUE=DATE:20260604
DTEND;VALUE=DATE:20260606
DTSTAMP:20260605T065404
CREATED:20260129T174306Z
LAST-MODIFIED:20260604T202522Z
UID:191911-1780531200-1780703999@www.bordeaux-neurocampus.fr
SUMMARY:14th Biology / Chemistry Young Scientists' Symposium
DESCRIPTION:Venue: IECB\, Bordeaux\n \n\nThe Young Scientists’ Symposium is a student-led scientific conference focused on the interface of chemistry and biology. Organized by PhD students from the University of Bordeaux\, this conference is open to all students and researchers interested in these fields\, regardless of their institutional affiliation. \nMore details on https://yss2026.sciencesconf.org/ \n
URL:https://www.bordeaux-neurocampus.fr/event/biology-chemistry-14th-young-scientists-symposium/
CATEGORIES:Hors Bordeaux Neurocampus,Symposiums
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20260605T093000
DTEND;TZID=Europe/Paris:20260605T093000
DTSTAMP:20260605T065404
CREATED:20260507T060104Z
LAST-MODIFIED:20260507T060104Z
UID:198091-1780651800-1780651800@www.bordeaux-neurocampus.fr
SUMMARY:ISVV seminar - Ardem Patapoutian
DESCRIPTION:Venue: Station Marne (16-22 cours de la Marne) \n\nArdem Patapoutian\nScripps Research Institute\nNobel Prize of medicine \nTitle\nPiezo ion channels as sensors for touch and beyond: Uncorking as possible role in mouthfeel \nRegistration\nhttps://evento.renater.fr/survey/conference-ardem-patapoutian-amphi-marne-5n83nhal \n
URL:https://www.bordeaux-neurocampus.fr/event/isvv-seminar-ardem-patapoutian/
CATEGORIES:A la une,Hors Bordeaux Neurocampus,Pour les scientifiques,Séminaire Impromptu
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20260605T120000
DTEND;TZID=Europe/Paris:20260605T120000
DTSTAMP:20260605T065404
CREATED:20260512T070406Z
LAST-MODIFIED:20260512T070849Z
UID:198231-1780660800-1780660800@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - Sarah Moyon
DESCRIPTION:Venue: Unité 1034 –\n1 avenue de Magellan – Pessac\nhttps://maps.app.goo.gl/neuEkruFjnJNmsu58 \n\nSarah Moyon\, PhD\nAix-Marseille University\, France\nhttps://www.linkedin.com/in/sarah-moyon-a7710619/\nhttps://inp.univ-amu.fr/fr/annuaire/sarah-moyon \nInvited by Candice Chapouly\nBiology of Cardiovascular Diseases\nhttps://u1034.bordeaux.inserm.fr/ \nTitle\nEpigenetic regulation of neuroglial communication in adaptive myelination and in age-related cognitive decline \nAbstract\nAging is associated with progressive cognitive decline\, partly linked to impaired myelin plasticity and adaptive myelination. Oligodendrocytes (OLs)\, the myelinating cells of the central nervous system\, support neuronal function through axonal insulation and metabolic coupling. With age\, myelin integrity and the capacity to generate new myelin decrease\, contributing to cognitive deficits. Emerging evidence indicates that intrinsic oligodendroglial alterations\, including epigenetic dysregulation\, underlie these defects. In particular\, downregulation of the epigenetic activator TET1 alters genes involved in neuroglial communication and the axon–myelin interface. \nWe have identified SLC12A2\, a Na+/K+/Cl- symporter localized at the neuroglial interface\, as downregulated in aged oligodendroglial cells. Reduced SLC12A2 expression is associated with structural abnormalities at the axon–myelin interface and impaired motor learning. Using an inducible oligodendrocyte-specific Slc12a2 knockout mouse model\, we show that SLC12A2 loss leads to motor learning deficits. In vitro\, Slc12a2-deficient OLs display reduced branching and decreased axon–myelin contact\, suggesting impaired differentiation and myelination. \nThis project aims to define how TET1-dependent regulation of SLC12A2 controls neuroglial communication\, thus myelin plasticity\, during aging and in age-related diseases. \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-sarah-moyon/
CATEGORIES:A la une,Hors Bordeaux Neurocampus,Pour les scientifiques
END:VEVENT
END:VCALENDAR