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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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TZID:Europe/Paris
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DTSTART:20260329T010000
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DTSTART:20261025T010000
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DTSTART;VALUE=DATE:20260519
DTEND;VALUE=DATE:20260522
DTSTAMP:20260406T052056
CREATED:20260303T090927Z
LAST-MODIFIED:20260303T225127Z
UID:195165-1779148800-1779407999@www.bordeaux-neurocampus.fr
SUMMARY:Journées du Réseau Français de la Recherche Reproductible
DESCRIPTION:Lieu : Campus Victoire\, Bordeaux \n\nLes journées du Réseau Français de la Recherche Reproductible (https://www.recherche-reproductible.fr/) auront lieu du 19 au 21 Mai 2026 à Bordeaux (site Victoire). Il s’agit d’un moment important pour la\nconsolidation du réseau et le dynamisme des collaborations émergentes. \nCe colloque fait suite aux journées organisées à Paris en 2023\, à Grenoble en 2024 et à Lyon en 2025. \nProgramme\nLa première journée sera consacrée à l’outil d’environnement logiciel reproductible Guix.\nLes deux journées suivantes seront quant-à-elles orientées vers des échanges multidiciplinaires autour de la reproductibilité avec des focus sur les SHS et la recherche expérimentale. \nProgramme complet \nInscription\nInscription gratuite mais obligatoire \n
URL:https://www.bordeaux-neurocampus.fr/event/journees-du-reseau-francais-de-la-recherche-reproductible/
CATEGORIES:A la une,IMN,Pour les scientifiques
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DTSTART;TZID=Europe/Paris:20260519T113000
DTEND;TZID=Europe/Paris:20260519T113000
DTSTAMP:20260406T052056
CREATED:20260327T134204Z
LAST-MODIFIED:20260401T123058Z
UID:196706-1779190200-1779190200@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - Anthony Hannan
DESCRIPTION:Venue: Centre Broca \n\nProf. Anthony J. Hannan\nFlorey Institute of Neuroscience and Mental Health\nUniversity of Melbourne\nhttps://florey.edu.au/researcher/anthony-hannan/ \nAnthonny Hannan is a visiting professor at LNEC (Poitiers) in May 2026 \nTitle\nGene-environment interactions modulating brain function within and between generations \nAbstract\nWe are interested in how gene-environment interactions mediate and modulate brain function\, in health and disease. We have examined the role of various molecular and cellular mediators\, and environmental modulators\, as they influence healthy cognitive and affective function\, as well as cognitive and affective disorders. Our findings have revealed key pathways implicated in the therapeutic impacts of environmental stimuli and identified novel therapeutic targets. We have also discovered altered brain-body interactions\, including the first evidence of gut dysbiosis (dysregulated gastrointestinal microbiota) in Huntington’s disease (where we model depression\, dementia and motor symptoms) and a genetic model of schizophrenia. We have found that environmental interventions which have therapeutic impacts also modulate the gut microbiome. Furthermore\, we have shown that a high-fibre diet and prebiotic interventions improved gut function and ameliorated cognitive and affective dysfunction. Ongoing studies are exploring the gut microbiome as a therapeutic target and the possibility that specific environmental factors may modulate brain function via microbiota-gut-brain interactions. Furthermore\, we are examining the neuroimmune impacts of the gut microbiome\, including area-specific changes in microglial activation states. These approaches to gene-environment interactions may facilitate the development of enviromimetics (including exercise mimetics as a subclass) for various brain disorders. \nIn order to understand how gene-environment interactions may sculpt brain development and function between generations\, we have also been exploring epigenetic inheritance via the paternal lineage. We have discovered transgenerational effects of various paternal environmental exposures\, including exercise\, stress\, diet\, microbiome modulation and immune activation. These lifestyle factors and experiences modulate sperm noncoding RNAs and alter development\, brain function and behaviour in offspring. We are exploring epigenetically mediated changes in the brains of offspring\, including microglial activation states and other forms of neuroimmune modulation. The effects on offspring phenotypes\, via epigenetic inheritance\, have relevance to the pathogenesis of various neuropsychiatric disorders\, including depression and anxiety disorders. \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-anthony-hannan/
CATEGORIES:A la une,Pour les scientifiques,Séminaire Impromptu
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