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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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DTSTART:20250330T010000
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DTSTART;TZID=Europe/Paris:20250523T090000
DTEND;TZID=Europe/Paris:20250523T170000
DTSTAMP:20260406T223119
CREATED:20250403T071431Z
LAST-MODIFIED:20250516T125751Z
UID:182540-1747990800-1748019600@www.bordeaux-neurocampus.fr
SUMMARY:Stand commercial Macherey-Nagel et Hamilton
DESCRIPTION:Lieu : centre Broca Nouvelle-Aquitaine \n\nMACHEREY-NAGEL \nMACHEREY-NAGEL est une société allemande renommée pour son expertise en biologie moléculaire. Depuis plus de 50 ans\, nous offrons des solutions innovantes et de haute qualité pour la purification de l’ADN\, de l’ARN et des protéines. Nos produits sont conçus pour répondre aux exigences des laboratoires de recherche et de diagnostic\, garantissant fiabilité et performance. \nNous serons ravis de discuter de vos besoins et de vous présenter nos produits et services en personne. \nHAMILTON ROBOTICS \nDepuis plus de 70 ans\, Hamilton Robotics travaille aux côtés de ceux qui sont à la pointe de la recherche. Cet échange de connaissances permet à Hamilton de transformer les dernières tendances scientifiques en solutions automatisées\, offrant ainsi aux scientifiques la technologie nécessaire pour accélérer leurs recherches. \nHamilton propose des stations de travail pour une large gamme d’applications courantes. Celles-ci comprennent l’extraction d’acides nucléiques\, la préparation de librairies NGS\, la PCR\, le fractionnement du sang\, la préparation d’échantillons LC-MS\, la culture cellulaire\, la culture des organoïdes et bien d’autres encore. \nL’ensemble de la gamme de produits offre un maximum de fiabilité\, d’efficacité\, de flexibilité et de convivialité. \n
URL:https://www.bordeaux-neurocampus.fr/event/stand-commercial-macherey-nagel-et-hamilton/
CATEGORIES:A la une,Pour les scientifiques,Stand ou séminaire commercial
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DTSTART;TZID=Europe/Paris:20250523T113000
DTEND;TZID=Europe/Paris:20250523T113000
DTSTAMP:20260406T223119
CREATED:20240710T170628Z
LAST-MODIFIED:20250428T091117Z
UID:173137-1747999800-1747999800@www.bordeaux-neurocampus.fr
SUMMARY:PhD seminar - Pauline Belujon
DESCRIPTION:Venue: Centre Broca \n\nPauline Belujon\nMCU\nLaboratoire de Neurosciences Expérimentales et Cliniques (LNEC)\, Poitiers\nhttps://lnec.labo.univ-poitiers.fr/membres/belujon-pauline/ \nTitle\nInsular Cortex and Dopamine System Dysregulation in the Addicted Brain \nAbstract\nMultiple lines of preclinical and clinical evidence have shown that prolonged exposure to drugs\, followed by abstinence\, induces lasting neuroadaptations across brain systems involved in affect regulation\, interoception\, stress response\, and cognitive control. These changes are characterized by disruptions in dopamine signaling\, particularly in the ventral tegmental area (VTA)\, where reduced dopaminergic activity impairs reward processing. Additionally\, alterations in synaptic plasticity in regions such as the nucleus accumbens and prefrontal cortex impact motivation\, emotion\, and decision-making. Our research focuses on understanding how chronic drug exposure and subsequent abstinence affect both the activity of dopamine neurons and synaptic plasticity within key brain circuits. A major focus of our work is the insular cortex\, a region critical for processing interoceptive and emotional information\, and increasingly recognized for its involvement in addiction. We explore how the insular cortex influences the activity of dopaminergic neurons in the VTA\, integrates inputs from other brain regions\, and modulates behavior during abstinence\, with a particular focus on how it contributes to the risk of relapse. By integrating electrophysiological techniques\, behavioral models of relapse\, and circuit-level analyses\, we aim to uncover how dysregulation of the dopamine system\, particularly within circuits involving the insular cortex\, contributes to persistent vulnerability during abstinence. Ultimately\, our research seeks to reveal how the insular cortex drives addiction-related brain changes and how this understanding can guide the development of targeted therapies for addiction recovery \nSelected publications\nGauthier M.\, Hebert LP.\, Dugast E.\, Lardeux V.\, Letort K\, Belnoue L\, Balado E\, Solinas M.\, Belujon P. (2025)\n« Sex-dependent effects of stress on aIC-NAc circuit neuroplasticity: Role of the endocannabinoid system”\nProg Neuropsychopharmacol Biol Psychiatry Mar 18:138:111335 \nSalin A.\, Dugast E.\, Lardeux V.\, Solinas M.\, Belujon P. (2023)\n“The amygdala-ventral pallidum pathway contributes to a hypodopaminergic state in the ventral tegmental area during protracted abstinence from chronic cocaine”\nBr J Pharmacol. 2023 Jul;180(14):1819-1831. \nSalin A.\, Lardeux V.\, Solinas M.\, Belujon P. (2021)\n“Protracted abstinence from extended cocaine self-administration is associated with hypodopaminergic activity in the VTA but not in the SNc”\nInt. J. Neuropsychopharmacol  Jul 14;24(6):499-504 \nNicolas C.\, Tauber C.\, Lepelletier F.X.\, Chalon S.\, Belujon P.\, Galineau L.\, Solinas M.  (2017)\n“Longitudinal changes in brain metabolic activity after withdrawal from escalation of cocaine self-administration”\nNeuropsychopharmacology Sep;42(10):1981-1990 \nBelujon P.\, Jakobowski N.L.\, Dollish H.K. and Grace A.A. (2016)\n“Withdrawal from acute amphetamine induces an amygdala-driven attenuation of dopamine neuron activity: reversal by ketamine”\nNeuropsychopharmacology. Jan;41(2):619-27 \nBelujon P. and Grace A.A. (2014)\n“Restoring balance in depression: ketamine reverses deficit in dopamine-dependent synaptic plasticity”\nBiological Psychiatry\, Dec 15;76(12):927-36 \n
URL:https://www.bordeaux-neurocampus.fr/event/phd-seminar-pauline-belujon/
CATEGORIES:A la une,Conférences mensuelles,INCIA,Pour les scientifiques
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