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X-WR-CALNAME:Bordeaux Neurocampus
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X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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DTSTART;VALUE=DATE:20241118
DTEND;VALUE=DATE:20241206
DTSTAMP:20260407T065734
CREATED:20240123T171300Z
LAST-MODIFIED:20241128T174622Z
UID:167216-1731888000-1733443199@www.bordeaux-neurocampus.fr
SUMMARY:Cajal lectures - Movement and motor control in health and disease
DESCRIPTION:The Brain Prize Course \nLectures are open to everyone. \nVenue: CARF \n\nNovember 18 – 11:00am\nSilvia Arber (Basel University\, Switzerland)\nDisentangling brainstem circuits for body movement. \nNovember 19 – 9:00am\nDavid McLean (University of Edinburgh\, UK)\nSizing up principles of recruitment during locomotion in zebrafish. \nNovember 20 – 9:00am\nSamuel Sober (Emory University\, USA)\nSpiking codes for skilled motor control. \nNovember 20 – 11:00am\nClaire Wyart (Paris Brain Institute\, France)\nOptical innovations to probe connectivity and functions of sensorimotor circuits of the brainstem and spinal cord. \nNovember 25 – 9:00am\nLora Sweeney (Institute of Science and Technology\, Austria)\nEvolution and development of swim versus limb motor circuits. \nNovember 25 – 11:00am\nJonathan Whitlock (KISN\, Norway)\nCortical integration of posture and active sensing in freely moving animals. \nNovember 28 – 9:00am\nCamille Jeunet (Bordeaux University\, France)\nBCI-based neurofeedback training procedures to restore or improve motor skills: a user-centred approach. \nNovember 28 – 11:00am\nMarie-Laure Welter (Paris Brain Institute\, France)\nUnderstanding Gait and Balance Disorders in Parkinson’s Disease: The Role of Basal Ganglia and Mesencephalic Locomotor Region Dysfunction. \nNovember 29 – 9:00am\nNicolas Mallet (Bordeaux University\, France)\nAbnormal Network Dynamics in Basal ganglia circuits during Parkinsonism: Origins and pathophysiological insights. \nDecember 2 – 9:00am\n Claire Meehan (University of Copenhagen\, Denmark)\nProbing intrinsic motoneurone excitability from mice to human. \nDecember 2 – 11:00am\nGilad Silberberg (Karolinska Institute\, Sweden)\nStriatal circuits underlying sensorimotor functions. \nDecember 3 – 9:00am\nIan Duguid (University of Edinburgh\, UK)\nNeural circuits for executing task-specific movements. \nDecember 3 – 11:00am\nRune Berg (University of Copenhagen\, Denmark)\nControl of movement by spinal circuitry: Model and population recordings. \nDecember 5 – 9:00am\nJoaquim Da Silva (Champalimaud CU\, Portugal)\nDirect and indirect pathway dynamics in dystonia. \nDecember 5 – 11:00am\nOle Kiehn (University of Copenhagen\, Denmark)\nUnraveling Brainstem Circuits for Movement: Insights into Motor Control and Implications for Treatment of Movement Disorders. \nMore details about the course\nWebsite: https://cajal-training.org/on-site/movement-and-motor-control-in-health-and-disease/ \n
URL:https://www.bordeaux-neurocampus.fr/event/cajal-lectures-the-brain-prize-course-movement-and-motor-control-in-health-and-disease/
CATEGORIES:A la une,Cajal Lectures,Pour les scientifiques
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20241126
DTEND;VALUE=DATE:20241127
DTSTAMP:20260407T065734
CREATED:20241108T135252Z
LAST-MODIFIED:20241108T135404Z
UID:177423-1732579200-1732665599@www.bordeaux-neurocampus.fr
SUMMARY:Chemistry Biology Interface Day 2024
DESCRIPTION:The Chemistry Biology Interface Day (CBID 2024) in Nouvelle Aquitaine will take place on November 26th at IECB (2 rue Escarpit\, 33600 Pessac). This scientific meeting is aiming at promoting research at the interface between chemistry and biology in the Nouvelle Aquitaine region. It is open to all\, from master students to researchers and will include a poster session and flash talks! Anyone working at this interface or interested in learning more is welcome to join! Please find the preliminary program at the end of this Email. \nPlease register to attend the symposium by November 15th 2024 using the link below. Attendance is free but required for logistic reasons as lunch will be provided. Please note that space is limited to 120 participants.\nhttps://framaforms.org/cbid2024-chemistry-biology-interface-day-1722509840 \nIf you’d like to present a poster\, please register and submit an abstract to e.thinon@iecb.u-bordeaux.fr by November 15th\, using the abstract template available on the registration page. Slots will also be available for flash poster presentations (selected from the abstracts). \nThis event is sponsored by the STS and SMR departments from the University of Bordeaux. \nThe organizing committee\nFrederic Friscourt (IECB/ISM) / Elisabeth Garanger (LCPO)/Jeanne Leblond-Chain (ARNA) / Nicolas Martin (CRPP) / Matthieu Sainlos (IINS) / Elia Stahl (IECB/LBM)/Emmanuelle Thinon (IECB/CBMN) \nMore details\, program\, registration\nhttps://sts.u-bordeaux.fr/evenements/cbid-2024 \n
URL:https://www.bordeaux-neurocampus.fr/event/chemistry-biology-interface-day-2024/
CATEGORIES:A la une,Pour les scientifiques,Symposiums
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20241126T103000
DTEND;TZID=Europe/Paris:20241126T103000
DTSTAMP:20260407T065734
CREATED:20241101T174032Z
LAST-MODIFIED:20250414T140815Z
UID:177048-1732617000-1732617000@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - Anne Louise Ponsonby
DESCRIPTION:Venue: Centre Broca \n \n\nAnne Louise Ponsonby\nFlorey Institute for Neuroscience and mental health – Melbourne\, Australia\nVisiting professor at the University of Bordeaux\nhttps://florey.edu.au/researcher/anne-louise-ponsonby/ \nInvited by Marc Landry (IMN) \nTitle\nPrenatal plastic chemical exposure\, molecular programming and offspring neurodevelopment \nBiosketch\nProfessor Anne-Louise Ponsonby (B Med Sci\, MBBS\, PhD\, FAFPHM\, FAFHMS\, RACP) is an epidemiologist and public health physician. Ponsonby is head of Neuroepidemiology of the Florey Institute of Neuroscience and Mental Health and a professor of Medicine at University of Melbourne. Ponsonby has extensive experience in the design\, conduct and analysis of population-based studies\, then public health translation. \nAbstract\nExposure to everyday environmental chemicals – e.g.\, chemicals in plastic products– during pregnancy may be harmful to brain development. One plausible mechanism for the male excess of autism spectrum disorder is via exposure to bisphenols and related chemicals that suppress cellular expression of the aromatase gene (CYP19A1)\, a key enzyme that converts androgens to estrogens in the developing brain. In the Barwon Infant Study (BIS) human birth cohort of 1\,074 children\, we show that higher prenatal maternal levels of BPA in the top quartile of prenatal maternal BPA levels were associated with higher autism spectrum ASD symptoms at age 2 and ASD diagnosis at age 9 only in males with low aromatase genetic pathway activity scores. Also\, higher prenatal bisphenol A (BPA) levels were predictive of higher methylation across the CYP19A1 brain promoter 1f region measured in the cord blood of offspring.These human epigenetic findings were also demonstrated in the Columbia Center for Children’s Environmental Health Mother and Newborn (CCCEH-MN) cohort. In tandem with these human studies\, we provide laboratory findings across structural\, functional\, transcriptomic and electrophysiological domains. Thus\, taken together\, we find that prenatal BPA exposure is associated with impaired brain aromatase function and ASD-related behaviors and brain abnormalities in males that may be reversible through postnatal intervention with 10HDA\, an estrogenic fatty acid. A human trial is being developed. \nWe have used the modern causal inference technique of molecular mediation to also investigate other underlying pathways between early environment plastic chemical exposure and adverse neurodevelopment\, highlighting the potential adverse role of prenatal inflammation\, immune disruption\, epigenetics and metabolomics. We find phthalate plastics associated with a maternal metabolic shift in pregnancy towards non-oxidative energy pathways\, which are inefficient compared to oxidative metabolism is associated with a higher risk of subsequent ASD symptoms and diagnosis. We investigate personalised prevention\, demonstrating children with certain genetic profiles such as predisposition for low antioxidant capacity are more vulnerable to the adverse effects of chemicals. Overall\, this work combining molecular biology and epidemiology aims to identify not only chemicals associated with more adverse child neurodevelopment outcomes\, but the underlying biological pathways that increase risk. \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-anne-louise-ponsonby/
CATEGORIES:A la une,IMN,Pour les scientifiques,Séminaire Impromptu
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