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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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DTSTART:20230326T010000
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DTSTART:20231029T010000
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20230916
DTEND;VALUE=DATE:20240617
DTSTAMP:20260423T000548
CREATED:20230831T131841Z
LAST-MODIFIED:20240529T183441Z
UID:162040-1694822400-1718582399@www.bordeaux-neurocampus.fr
SUMMARY:Exposition : Cervorama
DESCRIPTION:Agitez vos neurones ! \nA travers cette exposition\, Cap Sciences propose aux visiteurs de découvrir le cerveau sous toutes ses formes lors d’une visite ponctuée de manipulations\, de jeux et d’expériences… Ils pourront notamment explorer les mondes des cerveaux de l’escargot\, l’abeille\, le singe et l’homme\, tester leur mémoire dans le « cognitilab »\, découvrir leur cerveau en 3D grâce au cervomaton ou encore analyser les capacités des animaux ! \nUne exposition conçue et réalisée par Cap Sciences en partenariat avec Bordeaux Neurocampus\n \nEn savoir plus\nSite web : https://www.cap-sciences.net/au-programme/exposition/grand-public/cervorama/ \n
URL:https://www.bordeaux-neurocampus.fr/event/exposition-cervorama/
CATEGORIES:Evénements pour tous,not-calendar,pour tous homepage,Semaine du cerveau 2024
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20240403
DTEND;VALUE=DATE:20240420
DTSTAMP:20260423T000548
CREATED:20240123T164255Z
LAST-MODIFIED:20240419T132704Z
UID:167205-1712102400-1713571199@www.bordeaux-neurocampus.fr
SUMMARY:Cajal lectures - Optogenetics\, chemogenetics\, and biosensors for neural circuit research
DESCRIPTION:Venue: CGFB \nLectures are open to everyone. \n\nWednesday 3 April – 9:00am\nOfer Yihzar (Weizmann Institute of Science\, Israël)What is optogenetics good for? \nWednesday 3 April – 11:00am\nMichael Bruchas (University of Washington\, USA)\nDecoding the Neurobiological Mechanisms of Neuromodulation\n \nThursday 4 April – 9:00am\nJonas Wietek (Charité – Universitätsmedizin Berlin\, Germany)200 years of discovery\, 20 years of innovation: The Journey of Optogenetics and Rhodopsins. \nThursday 4 April – 11:00am\n Stefan Herlitze (Ruhr–University Bochum\, Germany)MoleOptogenetic control and visualization of GPCR pathways\, or a journey from mouse brain to bioluminescent and fluorescent fish. \nFriday 5 April – 9:00am\nSimon Wiegert (University of Heidelberg\, Germany)New lights on an old concept : investigating the link between pupil linked arousal neuromodulation. \nFriday5 April – 11:00am\nMarie Carlen (Karolinska Institute\, Sweden)\nWhat defines the prefrontal cortex? Large-scale spiking surveys outline the landscape of the mouse prefronal cortex.\n \nTuesday 9 April – 9:00am\nValentina Emiliani (Vision Institute\, France)Holographic all–optical manipulation of neuronal circuits. \nTuesday 9 April – 11:00am\nCyril Herry and Daniel Jercog (Bordeaux University\, France\, University of Copenhagen\, Denmark)\nFrom single cell to population analysis of defensive behaviors.\n \nSaturday 13 April – 9:00am\nLief Fenno (University of Texas\, USA)\nEngineered platforms for complex AAV cargo expression.\n \nSaturday 13 April – 11:00am\nYaniv Ziv (Weizmann Institute of Science\, Israël)\nRepresentational drift in the hippocampus and cortex. \nTuesday 16 April – 9:00am\nChristina Kim (UC Davis\, USA)Optogenetic reactivation of drug–ensembles using activity integrators. \nTuesday 16 April  – 11:00am\n Karl Deisseroth (Stanford University\, USA)Probing Mysteries of Brain Function: An Unexpected Journey Through the Inner Workings of Microbial Membrane Channels. \nFriday 19 April – 9:00am\nMackenzie Mathis (EPFL\, Swiss)\nMeasuring neural and behavioral dynamics.\n \nFriday 19 April – 11:00am\nAnna Beyeler (Bordeaux University\, France)\nLinking anxiety and emotional valence in circuits of the amygdala and insula.\n \n\nAbout the course\nWebsite: https://cajal-training.org/on-site/optogenetics-chemogenetics-biosensors/ \n
URL:https://www.bordeaux-neurocampus.fr/event/cajal-lectures-optogenetics-chemogenetics-and-biosensors-for-neural-circuit-research/
CATEGORIES:Cajal Lectures,Pour les scientifiques
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DTSTART;TZID=Europe/Paris:20240417T140000
DTEND;TZID=Europe/Paris:20240417T140000
DTSTAMP:20260423T000548
CREATED:20240305T122726Z
LAST-MODIFIED:20240410T115347Z
UID:169014-1713362400-1713362400@www.bordeaux-neurocampus.fr
SUMMARY:Seminar – Laura Bradfield
DESCRIPTION:Venue: Centre Broca \n\nDr. Laura Bradfield\nSenior Research Fellow\nHead of the Brain and Behaviour Lab\nSchool of Life Sciences\, UTS\, Sydney\, Australia \nInvited by Shauna Parkes (INCIA) \nTitle\nBehavioural and Brain Mechanisms of Sustained Contingency Degradation \nAbstract\nPreclinical models of treatments for substance use disorder have typically involved extinction\, during which once-rewarded cues or actions are no longer paired with reward. One problem with extinction\, however\, is that extinguished responding tends to spontaneously recover over time. We therefore tested whether contingency degradation might be effective in producing a reduction in responding that does not spontaneously recover. In Experiment 1\, rats were trained to press a left lever for pellets and a right lever for a sucrose solution\, or the opposite arrangement\, counterbalanced. Following training\, although both levers continued to earn their respective outcomes\, one of outcomes was also delivered when no lever press had occurred. This degraded the contingency between that lever and its outcome\, because rats selectively reduced responding on that lever. When given a 10 min test in which both levers were extended and no outcomes delivered\, rats responded in accordance with the degradation contingencies (i.e. NonDegraded > Degraded) regardless of whether they were tested 1 day or 2 weeks after degradation\, suggesting that degradation does not spontaneously recover. In Experiment 2\, we investigated whether chemogenetically inhibiting glutamatergic neurons in the lateral OFC impaired degradation learning using this paradigm. Animals injected with the control virus (mCitrine+DCZ group) learned degradation\, whereas animals for whom the lateral OFC was inhibited prior to degradation training (hM4Di+DCZ group) did not (i.e. nondegraded = degraded)\, and this pattern of results was retained at both immediate and delayed tests. A third experiment will determine whether this sustained impairment in extinction will persist when the lateral OFC is chemogenetically silenced during test. Together\, these experiments suggest that the behavioural and brain mechanisms of contingency degradation and extinction are distinct\, and that degradation is more effective in producing a lasting behavioural change. \nBiography\nDr Laura Bradfield is a Senior Research Fellow and head of the Brain and Behaviour Lab in the School of Life Sciences at UTS. She joined UTS as a Research Fellow in 2018 after undertaking her PhD and postgraduate studies at UNSW Sydney. Laura’s primary areas of interest include investigating the behavioural and brain mechanisms of compulsive disorders\, the glial mechanisms of goal-directed decision-making\, and contextual modulation of goal-directed action. Her work is currently funded by two NHMRC Ideas Grants\, and she has previously been awarded an ARC Discovery Project as well as two NHMRC project grants. In total Laura’s research has attracted approximately $3.5 million. Laura has published 30+ journal articles and three book chapters\, including first author articles in the influential journals Neuron and Nature Neuroscience. She is a Reviewing Editor at eLife\, eNeuro and Frontiers in Behavioural Neuroscience\, and an Associate Editor at the Journal of Neuroscience. Laura has spoken widely about her research at conferences and events in Australia and overseas. In 2022\, she won the UTS Faculty of Science’s “Supervisor of the Year” award\, and she is also passionate about increasing diversity in science and improving conditions within the scientific community for underrepresented individuals. \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-laura-bradfield/
CATEGORIES:A la une,Pour les scientifiques,Séminaire Impromptu
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