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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr
X-WR-CALDESC:Évènements pour Bordeaux Neurocampus
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TZID:Europe/Paris
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DTSTART:20220327T010000
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DTSTART:20221030T010000
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DTSTART;VALUE=DATE:20220211
DTEND;VALUE=DATE:20230103
DTSTAMP:20260423T000804
CREATED:20220209T143152Z
LAST-MODIFIED:20220218T181852Z
UID:144814-1644537600-1672703999@www.bordeaux-neurocampus.fr
SUMMARY:Exposition : "Cell Immersion"
DESCRIPTION:Les Bassins des Lumières – Imp. Brown de Colstoun\, Bordeaux \n\nCell Immersion vous convie à un voyage Art & Science dans un monde microscopique méconnu : l’humain. \nL’œuvre est la première brique d’un projet d’envergure\, nommé Cell Worlds\, qui amène les images de microscopie là où elles ne sont jamais allées. Loin des laboratoires et des disques durs des scientifiques et au plus près des cellules humaines. Ici\, tout est bien réel\, et surtout vivant. Chaque visuel de l’exposition se compose de véritables cellules : de l’électrisant neurone au fragile embryon en passant par les mélancoliques flux sanguins du cerveau. Découvrez un univers aux couleurs chatoyantes et d’une diversité incroyable. \nCell Immersion est une première mondiale scientifique\, mettant en scène le vivant microscopique dans des proportions jamais tentées. C’est également un des plus grands showcases de la recherche scientifique\, regroupant de nombreuses équipes\, laboratoires et instituts du monde entier. Une preuve que la science et l’art n’ont pas de frontières. \nÀ travers l’émerveillement\, ce voyage saura éveiller votre curiosité scientifique. Au-delà du simple divertissement\, Cell Immersion vous invite à vous reconnecter avec le monde vivant microscopique qui est aujourd’hui trop inconnu\, trop peu contemplé et parfois trop mystifié. \n  \n\nRéalisation : Terence Saulnier et Renaud Pourpre \nComposition de la bande originale : Youenn Lerb \n– \nEntrée gratuite dans la limite des places disponibles\nPasse vaccinal et masque obligatoires \n\n  \nPour plus d’informations : https://www.bassins-lumieres.com/fr/cell-immersion \n
URL:https://www.bordeaux-neurocampus.fr/event/exposition-cell-immersion/
LOCATION:Les Bassins des Lumières\, Imp. Brown de Colstoun\, Bordeaux\, 33000\, France
CATEGORIES:not-calendar,Semaine du cerveau 2022
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DTSTART;TZID=Europe/Paris:20221104T100000
DTEND;TZID=Europe/Paris:20221104T100000
DTSTAMP:20260423T000804
CREATED:20220921T084258Z
LAST-MODIFIED:20220921T134747Z
UID:151047-1667556000-1667556000@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - Raphael Lamprecht
DESCRIPTION:Raphael Lamprecht\nSalle Purkinje (Centre Broca Nouvelle-Aquitaine) \n\nRaphael Lamprecht\nSagol Department of Neurobiology\, University of Haifa\, Haifa\, Israel \nInvited by Daniel Choquet (IINS) \nTitle\nThe roles of EphB2 and ephrinB2 in long-term memory formation \nAbstract\nEph receptors and their cognate ephrin ligands can signal in a bidirectional fashion between two opposing cells. They are attractive candidates to play a central role in memory formation as they are involved in the regulation of synaptic transmission and neuronal morphogenesis. We aim to explore the roles of EphB2 and ephrinB2 signaling in memory formation and enhancement. We found that activation of EphB2\, using a novel technology termed optoEphB2 that allows the control of EphB2 forward signaling activity by light\, enhances memory and that mice that express EphB2 that lacks forward signaling are impaired in the ability to form long-term memory. To explore the roles of ephrinB2 in memory we depleted ephrinB2 in different cell populations in the brain and studied the effect on the ability of the mice to form long-term memory. We revealed that ephrinB2 in different cell populations is essential for long-term memory formation. We also provide evidence for downstream molecular signaling pathways that can mediate EphB2 and ephrinB2 functions in memory formation. \n
URL:https://www.bordeaux-neurocampus.fr/event/seminar-raphael-lamprecht/
CATEGORIES:A la une,Pour les scientifiques,Séminaire Impromptu
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DTSTART;TZID=Europe/Paris:20221104T113000
DTEND;TZID=Europe/Paris:20221104T123000
DTSTAMP:20260423T000804
CREATED:20211110T170812Z
LAST-MODIFIED:20221028T143932Z
UID:140919-1667561400-1667565000@www.bordeaux-neurocampus.fr
SUMMARY:Monthly conference (PhD seminar) - Marion Leboyer
DESCRIPTION: \nVenue: Centre Broca Nouvelle-Aquitaine \n\nMarion Leboyer\nUniversité Paris-Est-Créteil\nDepartment of Addictology and Psychiatry (Mondor hospital\, AP-HP)\nTranslational Neuro-Psychiatry Laboratory (Inserm U955)\nfoundation FondaMental \nInvited by Yoni Couderc (PhD student – Beyeler’s tream – Magendie) \nTitle\n Immuno-Inflammation of Psychiatric disorders \nAbstract\nThe discovery that the immune system can influence brain function and structure has profoundly changed the landscape of psychiatry. Repeated report of association of pro-inflammatory cytokines with major psychiatric disorders led to exploration of the causes and consequences of this inflammatory background.  This low-grade inflammation has been shown to be the consequence of interaction between environmental factors such as infections\, stress\, pollution\, unhealthy life style with immune-genetic background. Association with particular immune-genetic variants of Toll-like receptor genes possibly explain diminished response to infections (TLR\, NOD)\, association with mitochondrial genes contribute to maintenance of inflammation\, while association with particular HLA haplotypes explains induction of auto-immune phenomena and/or exaggerated synaptic pruning.  For example\, association with the complement genes can induce abnormal pruning  and microglial activation thereby increasing the risk of neurodevelopmental disorders such as early onset schizophrenia. In the context of the SARS-Cov2 pandemic\, increased severity of COVID-19 in psychiatric patients is probably due to their reduced ability to fight infection. Systemic inflammation and persistent infections induce different pathways paving the way to biomarker-guided personalized medicine. One of the best example is the identification of “autoimmune psychosis” defined by presence of anti-neuronal antibodies that has been confounded for long with atypical\, mild\, or attenuated forms of autoimmune encephalitis. Persistent infections are associated to activation of Human endogenous retrovirus (HERV). Systemic inflammation induced by microbial infection or psychosocial factors can also be at the origin of the activation of human endogenous retrovirus. Inflammation is also known to be associated with gut dysbiosis and disturbance of the integrity of the digestive barrier leading to behavioral abnormalities. One last example can be found in the immune-metabolic abnormalities that pave the way to metabolic syndrome associated with psychiatric disorders. Although many aspects of the complex relationship between immunity and brain function are not yet fully elucidated\, the findings that have accumulated so far have transformed our understanding of psychiatric disorders and favored the consideration of other possible cellular and molecular targets for their treatment than just alterations in neuronal transmitters. \n  \n\nPhD seminars are organized by the NBA\, Bordeaux Neurocampus\, and the Bordeaux Neurocampus Graduate Program \n
URL:https://www.bordeaux-neurocampus.fr/event/monthly-conference-november-2022/
CATEGORIES:A la une,Conférences mensuelles,Pour les scientifiques
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