1.0Bordeaux Neurocampushttps://www.bordeaux-neurocampus.fr/en/Abrous D. Norahttps://www.bordeaux-neurocampus.fr/en/author/nora-abrousinserm-fr/Memory - Bordeaux Neurocampusrich600338<blockquote class="wp-embedded-content" data-secret="WxFwVG3e5o"><a href="https://www.bordeaux-neurocampus.fr/en/article/memory/">Memory</a></blockquote><iframe sandbox="allow-scripts" security="restricted" src="https://www.bordeaux-neurocampus.fr/en/article/memory/embed/#?secret=WxFwVG3e5o" width="600" height="338" title="“Memory” — Bordeaux Neurocampus" data-secret="WxFwVG3e5o" frameborder="0" marginwidth="0" marginheight="0" scrolling="no" class="wp-embedded-content"></iframe><script type="text/javascript">
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1. Cell Mol Life Sci. 2006 May;63(9):999-1008. Mechanisms of memory stabilization and de-stabilization. Alberini CM(1), Milekic MH, Tronel S. Author information: (1)Department of Neuroscience, Mount Sinai School of Medicine, Box 1065, New York, NY 10029, USA. Cristina.Alberini@mssm.edu Memories become stabilized through a time-dependent process that requires gene expression and is commonly known as consolidation. During this time, memories are labile and can be disrupted by a number of interfering events, including electroconvulsive shock, trauma and other learning or the transient effect of drugs such as protein synthesis inhibitors. Once consolidated, memories are insensitive to these disruptions. However, they can again…