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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr/en/
X-WR-CALDESC:Events for Bordeaux Neurocampus
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TZID:Europe/Paris
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TZNAME:CEST
DTSTART:20250330T010000
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DTSTART:20251026T010000
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20251013
DTEND;VALUE=DATE:20251017
DTSTAMP:20260406T232450
CREATED:20250425T144544Z
LAST-MODIFIED:20251016T134806Z
UID:183448-1760313600-1760659199@www.bordeaux-neurocampus.fr
SUMMARY:International Society for Tractography Conference
DESCRIPTION:Venue: Centre Broca \n\nThis unique event will bring together 150 participants in the field of tractography\, focusing on anatomy\, methodologies\, and applications in both clinical and scientific contexts. With an innovative conference format designed to foster knowledge sharing and collaboration\, the conference – aligned with the International Society for Tractography’s raison d’être – aims to advance research and innovation in our common field of interest\, tractography. \nThe mornings will feature keynote lectures\, short oral presentations\, and poster sessions\, while the afternoons will be dedicated to interactive workshops tackling key challenges in tractography. \nMore details \n
URL:https://www.bordeaux-neurocampus.fr/en/event/international-society-for-tractography-conference/
CATEGORIES:For scientists,IMN,Symposium
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DTSTART;TZID=Europe/Paris:20251013T113000
DTEND;TZID=Europe/Paris:20251013T113000
DTSTAMP:20260406T232450
CREATED:20250923T133114Z
LAST-MODIFIED:20250923T141200Z
UID:188218-1760355000-1760355000@www.bordeaux-neurocampus.fr
SUMMARY:Seminar - Roberto De Pasquale
DESCRIPTION:Venue : CARF  \n\nRoberto De Pasquale\nDepartment of Physiology and Biophysics\, São Paulo \nInvited by Jérôme Baufreton (IMN) \nTitle\nThe effects of caffeine on synaptic plasticity and memory in the aging brain \nAbstract\nCaffeine consumption is believed to offer protective effects against neurodegeneration associated with aging by decreasing the production of reactive oxygen species and beta-amyloid peptide. Furthermore\, caffeine may enhance cognitive processes by influencing the mechanisms of synaptic plasticity that are fundamental to memory. Specifically\, in the hippocampus\, caffeine has the capacity to change the modulation of plasticity governed by adenosine receptors. Several studies have indicated that chronic caffeine intake can restore long-term potentiation (LTP) in the hippocampus under pathological conditions. Drawing from these findings\, we examined the effects of chronic caffeine consumption on memory and hippocampal synaptic transmission in SAMP8 animals\, which serve as a model for accelerated aging and neurodegeneration. We employed behavioral tests to assess recognition memory and we applied in vitro electrophysiology techniques to evaluate excitability and synaptic transmission in the hippocampus. Our results demonstrate that chronic caffeine consumption diminishes enhances hippocampal-dependent memory\, and facilitates LTP in hippocampal synapses. \nPublications\n5-HT-dependent synaptic plasticity of the prefrontal cortex in postnatal development. Guilherme Shigueto Vilar Higa\, José Francis-Oliveira\, Estevão Carlos-Lima\, Alicia Moraes Tamais\, Fernando da Silva Borges\, Alexandre Hiroaki Kihara\, Ianê Carvalho Shieh\, Henning Ulrich\, Silvana Chiavegatto\, Roberto De Pasquale. Sci Rep. 2022 Dec 5;12(1):21015. doi: 10.1038/s41598-022-23767-9. \nRegulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Guilherme Juvenal\, Guilherme Shigueto Vilar Higa\, Lucas Bonfim Marques # 1\, Thais Tessari Zampieri\, Felipe José Costa Viana\, Luiz R Britto\, Yong Tang\, Peter Illes\, Francesco di Virgilio \, Henning Ulrich\, Roberto de Pasquale Purinergic Signal. 2025 Feb;21(1):149-177. doi: 10.1007/s11302-024-10034-x. Epub 2024 Jul 24. \nSerotonergic neuromodulation of synaptic plasticity. Guilherme Shigueto Vilar Higa 1\, Felipe José Costa Viana 2\, José Francis-Oliveira 3\, Emily Cruvinel 2\, Thainá Soares Franchin 2\, Tania Marcourakis 4\, Henning Ulrich 5\, Roberto De Pasquale 6 Neuropharmacology. 2024 Oct 1:257:110036. doi: 10.1016/j.neuropharm.2024.110036. Epub 2024 Jun 12. \nHippocampal Interneurons Shape Spatial Coding Alterations in Neurological Disorders. Juliane Midori Ikebara # 1 2\, Renata Silva Jorge # 1\, Luciana Simões. Rafagnin Marinho 1\, Guilherme Shigueto Vilar Higa 1 3\, Avishek Adhikari 4\, Fernando M C V Reis 5\, Fernando S Borges 1 6\, Henning Ulrich 3\, Silvia Honda Takada 1\, Roberto De Pasquale 7\, Alexandre Hiroaki Kihara 8. Mol Neurobiol. 2025 May 20. doi: 10.1007/s12035-025-05020-2. Online ahead of print. \n
URL:https://www.bordeaux-neurocampus.fr/en/event/seminar-roberto-de-pasquale/
CATEGORIES:For scientists,home-event,IMN,Impromptu seminar
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BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20251013T140000
DTEND;TZID=Europe/Paris:20251013T140000
DTSTAMP:20260406T232450
CREATED:20251009T105820Z
LAST-MODIFIED:20251009T105820Z
UID:188675-1760364000-1760364000@www.bordeaux-neurocampus.fr
SUMMARY:CBMN seminar - Marina Casiraghi
DESCRIPTION:Venue:  IECB \n\nTitle\nStructural and dynamic insights into G protein–coupled receptor signaling \nAbstract\nG protein coupled receptors (GPCRs) form the largest family of membrane proteins. They mediate most\nresponses to hormones and neurotransmitters and underlie the senses of sight\, smell and taste. Nearly\nhalf of all approved drugs act on GPCRs\, which are also implicated in conditions such as drug addiction\,\nincluding opioid dependence. Despite their structural diversity (~800 members) GPCRs couple to only four\nG protein subfamilies. The molecular determinants underlying GPCR selectivity remain poorly understood\,\nyet such knowledge is essential for developing drugs that preferentially activate selected signaling\npathway\, a paradigm known as biased agonism. The β₂-adrenergic receptor (β₂AR)\, a prototypical class A\nGPCR\, has been used to characterize GPCR selectivity and biased agonism through a combination of\ncomplementary structural (cryo-EM)\, biochemical and biophysical methods (EPR\, fluorescence\nspectroscopy\, single-molecule FRET). Our data provide mechanistic insights into G-protein subtype\nselectivity at the β₂AR and the molecular basis of GPCR activation and signaling in response to biased\nligands. \nBiography\nDr. Casiraghi received her master’s degree in molecular biology from University of Milano Bicocca\, Italy.\nShe conducted her PhD at the Institut de biologie physico-chimique (IBPC) in Paris under the supervision\nof Dr. Laurent Catoire\, focusing on the investigation of G-protein coupled receptors (GPCRs)\nconformational landscape by solution-state NMR. For her postdoctoral work\, she joined the laboratory of\nProf. Brian K. Kobilka at Stanford University\, USA. At Stanford\, she bridged structural determination by\ncryo-EM and biophysical investigations to address the molecular determinants of GPCR activation and\nsignaling. While at Stanford\, Dr. Casiraghi was the recipient of the Marie Sklodowska-Curie postdoctoral\nfellowship and American Heart Association (AHA) postdoctoral fellowship. Currently\, she is a Young\nResearchers Marie Sklodowska-Curie fellow at University of Milan\, Italy\, where her research focuses on\nnovel therapeutic strategies to target GPCRs implicated in addiction. \n
URL:https://www.bordeaux-neurocampus.fr/en/event/cbmn-seminar-by-marina-casiraghi/
CATEGORIES:For scientists,home-event,Impromptu seminar
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