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DTSTART;TZID=Europe/Paris:20240209T100000
DTEND;TZID=Europe/Paris:20240209T123000
DTSTAMP:20260410T104353
CREATED:20230505T103801Z
LAST-MODIFIED:20240130T160039Z
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SUMMARY:Monthly conference (PhD seminar) - Tara Spires-Jones and Violetta Zujovic
DESCRIPTION:Venue: Centre Broca \n\nIn the frame of the International Day of Women and Girls in Science (February 11th) \n10:00 – Violetta Zujovic\nParis Brain Institute\nhttps://institutducerveau-icm.org/en/member/?user=1265 \nA neuroscientific approach to reach gender equity \nAbstract: Despite the increasing acceptance of egalitarian gender norms in democratic societies\, a striking contrast persists in the current societal reality. One of the main reasons gender inequalities persist is the perpetuation of implicit biases and gender stereotypes. These shape the way we behave\, our ability to recognize unequal treatment and the willingness of disadvantaged individuals to speak up. The neuroscience community is at the forefront of not only raising awareness of these unconscious biases\, but also has the tools to understand their cognitive origins and thus the potential to break down social stereotypes. \n\n10:30 – Coffee break \n11:20 – Marian Diamond Prize announcement\n11:30 – Tara Spires-Jones\nProf. Tara Spires-Jones\nUniversity of Edinburgh\nhttps://discovery-brain-sciences.ed.ac.uk/ \nIntroduction about her journey in Neuroscience as a woman \nImaging synapses in Alzheimer’s disease and related dementias \nSynapse loss correlates strongly with the progression of symptoms in Alzheimer’s disease (AD) and other forms of dementia\, but mechanisms of synapse loss remain poorly understood.  Our group has observed the accumulation of pathological proteins amyloid beta\, tau\, and alpha synuclein within individual synapses in neurodegenerative diseases. Further\, we find evidence of trans-synaptic spread of pathological proteins from pre to post synapses which contributes to the spread of pathology and neurodegeneration through the brain.  We also observe that astrocytes and microglia are important in synapse degeneration. Understanding synapse degeneration and the spread of pathological proteins through synapses will be important for developing disease modifying therapeutics for Alzheimer’s disease and other neurodegenerative disorders. \nBio \nTara Spires-Jones’ research focuses on the mechanisms and reversibility of neurodegeneration in Alzheimer’s disease\, other degenerative brain diseases\, and ageing.   With a vibrant team of researchers\, her work has shown that soluble forms of the pathological proteins amyloid beta and tau contribute to synapse and neurodegeneration\, and that lowering levels of these proteins can ameliorate phenotypes in model systems. Her group has also discovered that pathological forms of tau spread through the brain via synaptic connections. Further\, she has pioneered high-resolution imaging techniques in human post-mortem brain and found evidence that pathological proteins accumulate in synapses in human disease. \nIn addition to research\, Prof Spires-Jones is passionate about communicating scientific findings to the public and policy makers; increasing the rigour and reproducibility in translational neuroscience; promoting inclusivity and diversity in science; and supporting career development of neuroscientists.  She is President of the British Neuroscience Association (2023-2025). \nSelected publications \nColom-Cadena\, M.\, Davies\, C.\, Sirisi\, S.\, Lee\, J.-E.\, Simzer\, E. M.\, Tzioras\, M.\, Querol-Vilaseca\, M.\, Sánchez-Aced\, É.\, Chang\, Y. Y.\, Holt\, K.\, McGeachan\, R. I.\, Rose\, J.\, Tulloch\, J.\, Wilkins\, L.\, Smith\, C.\, Andrian\, T.\, Belbin\, O.\, Pujals\, S.\, Horrocks\, M. H.\, … Spires-Jones\, T. L. (2023). Synaptic oligomeric tau in Alzheimer’s disease—A potential culprit in the spread of tau pathology through the brain. Neuron\, 111(14)\, 2170-2183.e6. https://doi.org/10.1016/j.neuron.2023.04.020 \nKing\, D.\, Holt\, K.\, Toombs\, J.\, He\, X.\, Dando\, O.\, Okely\, J. A.\, Tzioras\, M.\, Rose\, J.\, Gunn\, C.\, Correia\, A.\, Montero\, C.\, McAlister\, H.\, Tulloch\, J.\, Lamont\, D.\, Taylor\, A. M.\, Harris\, S. E.\, Redmond\, P.\, Cox\, S. R.\, Henstridge\, C. M.\, … Spires-Jones\, T. L. (2023). Synaptic resilience is associated with maintained cognition during ageing. Alzheimer’s & Dementia: The Journal of the Alzheimer’s Association\, 19(6)\, 2560–2574. https://doi.org/10.1002/alz.12894Tzioras\, M.\, Daniels\, M. J. D.\, Davies\, C.\, Baxter\, P.\, King\, D.\, McKay\, S.\, Varga\, B.\, Popovic\, K.\, Hernandez\, M.\, Stevenson\, A. J.\, Barrington\, J.\, Drinkwater\, E.\, Borella\, J.\, Holloway\, R. K.\, Tulloch\, J.\, Moss\, J.\, Latta\, C.\, Kandasamy\, J.\, Sokol\, D.\, … Spires-Jones\, T. L. (2023). Human astrocytes and microglia show augmented ingestion of synapses in Alzheimer’s disease via MFG-E8. Cell Reports Medicine\, 101175. https://doi.org/10.1016/j.xcrm.2023.101175 \nTzioras\, M.\, McGeachan\, R. I.\, Durrant\, C. S.\, & Spires-Jones\, T. L. (2023). Synaptic degeneration in Alzheimer disease. Nature Reviews. Neurology\, 19(1)\, 19–38. https://doi.org/10.1038/s41582-022-00749-z \nPickett\, E. K.\, Herrmann\, A. G.\, McQueen\, J.\, Abt\, K.\, Dando\, O.\, Tulloch\, J.\, Jain\, P.\, Dunnett\, S.\, Sohrabi\, S.\, Fjeldstad\, M. P.\, Calkin\, W.\, Murison\, L.\, Jackson\, R. J.\, Tzioras\, M.\, Stevenson\, A.\, d’Orange\, M.\, Hooley\, M.\, Davies\, C.\, Colom-Cadena\, M.\, … Spires-Jones\, T. L. (2019). Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer’s Disease. Cell Reports\, 29(11)\, 3592-3604.e5. https://doi.org/10.1016/j.celrep.2019.11.044 \nColom-Cadena\, M.\, Pegueroles\, J.\, Herrmann\, A. G.\, Henstridge\, C. M.\, Muñoz\, L.\, Querol-Vilaseca\, M.\, Martín-Paniello\, C. S.\, Luque-Cabecerans\, J.\, Clarimon\, J.\, Belbin\, O.\, Núñez-Llaves\, R.\, Blesa\, R.\, Smith\, C.\, McKenzie\, C.-A.\, Frosch\, M. P.\, Roe\, A.\, Fortea\, J.\, Andilla\, J.\, Loza-Alvarez\, P.\, … Lleó\, A. (2017). Synaptic phosphorylated α-synuclein in dementia with Lewy bodies. Brain : A Journal of Neurology\, 140(12)\, 3204–3214. https://doi.org/10.1093/brain/awx275 \n  \n12:30 – Pizza lunch with both speakers and Doctoral + Postdoctoral researchers\n/ On registration until February 6th\n\nPhD seminars are organized by the NBA\, Bordeaux Neurocampus\, and the Bordeaux Neurocampus Graduate Program. \nThis special edition is coorganized by the Neurocampus Parity Commitee \n
URL:https://www.bordeaux-neurocampus.fr/en/event/phd-seminar-february2024/
CATEGORIES:For scientists,home-event,Monthly conferences
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BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20240209T112000
DTEND;TZID=Europe/Paris:20240209T112000
DTSTAMP:20260410T104353
CREATED:20240118T115824Z
LAST-MODIFIED:20240118T122149Z
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SUMMARY:Marian Diamond Prize: awardee announcement
DESCRIPTION:Atrium du centre Broca Nouvelle-Aquitaine \nAbout the prize\nBordeaux Neurocampus launches its 1st call to recognize outstanding achievements in Neuroscience of an early career female researcher in our community. \nMarian Diamond\nMarian Diamond (1926-2017) was an American pioneer in anatomical neuroscience who provided the first scientific evidence for anatomical neuroplasticity in the early 1960s. Later\, her work on Einstein’s brain in the 1980s gave new impetus to the study of neuron-glia interactions. \nInitiated by the Neurocampus Parity Committee (NPC)\, the Marian Diamond Prize aims to raise the profile of female neuroscientists at a crucial stage in their careers\, the postdoctoral period\, when they face many barriers to reach academic positions. \nThe winner will receive a cash prize of €1000\, and will be invited to present her research to the whole community on May 16th 2024 at the Bordeaux Neurocampus Day. \nMore details: text of the call \n  \n
URL:https://www.bordeaux-neurocampus.fr/en/event/marian-diamond-prize-awardee-announcement/
CATEGORIES:For scientists,home-event
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