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X-WR-CALNAME:Bordeaux Neurocampus
X-ORIGINAL-URL:https://www.bordeaux-neurocampus.fr/en/
X-WR-CALDESC:Events for Bordeaux Neurocampus
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DTSTART:20250330T010000
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DTSTART:20251026T010000
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20250922
DTEND;VALUE=DATE:20251011
DTSTAMP:20260526T142521
CREATED:20250221T135021Z
LAST-MODIFIED:20251009T092733Z
UID:181026-1758499200-1760140799@www.bordeaux-neurocampus.fr
SUMMARY:Cajal lectures: "Cancer and Neuroscience"
DESCRIPTION:Venue : CARF \nOrganized by the Bordeaux School of Neuroscience. \nLectures are open to everyone. \n\n\n\nProgramme\n\n\n\n\nMonday 22 September – 11:00am\nAnna Golebiewska (Luxembourg Institute of Health\, Luxembourg)\nHeterogeneity and plasticity of the brain tumor ecosystem: What can we learn from advanced patient-derived models? \nTuesday 23 September – 9:00am\nSimona Parrinello (University College London\, UK)\nInjury programmes shape gliomagenesis. \nTuesday 23 September – 11:00am\nHenrik Heiland (Dep. of Neurosurgery\, University Clinic Erlangen\, Germany)\nTBA\n \nThursday 25 September – 9:00am\n Hélène Castel (Cancer and Brain Genomics\, Inserm\, University of Rouen\, France)\nNeurosciences in Oncology: When Cancer Thinks Out of the Box. \nThursday 25 September – 11:00am \nThomas Daubon (CNRS\, University of Bordeaux\, France)\nLactate Shuttle between Glioblastoma Cells and Neurons: a Cancer Neuroscience Case. \nMonday 29 September – 9:00am\nFranck Winkler (University of Heidelberg\, Germany)\nCancer Neuroscience of Brain Tumors. \nMonday 29 September – 11:00am \nVidhya M. Ravi (Medical Centre University Clinic of Freiburg\, Germany)\nTumor–Host Interdependence: Decoding the Microenvironmental Dialogue. \nThursday 2 October – 9:00am \nHrvoye Miletic (University of Bergen\, Norway)\nCell communication and mitochondria transfer in brain tumors. \nThursday 2 October – 11:00am\nManuel Valiente (Spanish National Cancer Research Centre\, Spain)\nThe Evolving Landscape of Brain Metastases. \nFriday 3 October – 9:00am\nLeila Akkari (Netherlands Cancer Institute\, The Netherlands)\nMacrophages: Jack of all Trades in the Brain Ecosystem? \nMonday 6 October – 9:00am\nVarun Venkataramani (German Cancer Research Center\, Germany)\nTBA \nOctober 6 – 11:00am\nAurélie Tchoghandjian (Institute of Neurophysiopathology\, Marseille\, France)\nGlioblastoma stemness and immunosuppression: breaking the lethal cycle. \nOctober 9 – 9:00am\nAntonio Pagano Zottola (Bordeaux Institute of Oncology\, University of Bordeaux\, France)\nMitoDREADD: a new mitochondria-specific chemogenetic tool. \n\n\n\nCourse Directors \n\nAnna Golebiewska – Luxembourg Institute of Health\, Luxembourg\nAntonio C. Pagano Zottola – Bordeaux Institute of Oncology – Inserm\, France\nVarun Venkataramani – Universität Heidelberg\, Germany\n\n\nMore details about the course \n
URL:https://www.bordeaux-neurocampus.fr/en/event/cajal-lectures-cancer-and-neuroscience/
CATEGORIES:Cajal Lectures,For scientists
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20251006
DTEND;VALUE=DATE:20251010
DTSTAMP:20260526T142521
CREATED:20241016T084847Z
LAST-MODIFIED:20251009T092654Z
UID:177041-1759708800-1760054399@www.bordeaux-neurocampus.fr
SUMMARY:FINS 2025
DESCRIPTION:Venue: Centre Broca \n\nCo-organised by the Interdisciplinary Institute for Neuroscience (IINS) and the Bordeaux and Quebec Imaging Centers\, the Frontiers In Neurophotonics Symposium (FINS) 2025 will be the 7th in a biennial series of conferences dedicated to the new frontiers of microscopy and neuroscience. FINS brings together world experts to discuss and debate new developments in the field and how they can be used to study brain function. The world’s leading experts in the field of neuroimaging (including Nobel Prize winners) attend this biennial event.\nPrevious editions have alternated between Bordeaux (2009\, 2013\, 2017) and Quebec City (2011\, 2015\, 2022)\, attracting around 150 participants from all over the world. \nScientific committee:\n \n\nAnna Brachet (Bordeaux)\nDaniel Choquet (Bordeaux)\nLaurent Cognet (Bordeaux)\nPaul De Koninck (Laval)\nYves De Koninck (Laval)\nMathieu Ducros (Bordeaux)\nFlavie Lavoie-Cardinal (Laval)\nMarie-Eve Paquet (Laval)\nJean-Baptiste Sibarita (Bordeaux)\n\nAll details on: https://frontiersneurophotonics.org/ \nDonwload the Abstract Book FINS 2025 \n
URL:https://www.bordeaux-neurocampus.fr/en/event/fins-2025/
CATEGORIES:For scientists,IINS,Symposium
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BEGIN:VEVENT
DTSTART;VALUE=DATE:20251006
DTEND;VALUE=DATE:20251011
DTSTAMP:20260526T142521
CREATED:20250425T164326Z
LAST-MODIFIED:20251001T092950Z
UID:183450-1759708800-1760140799@www.bordeaux-neurocampus.fr
SUMMARY:Neuroscience Alliance Workshop
DESCRIPTION:Venue: Bordeaux \n\nEmergent Properties of the Connected Brain \nMore details \n  \n
URL:https://www.bordeaux-neurocampus.fr/en/event/neuroscience-alliance-workshop/
CATEGORIES:For scientists,Symposium
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20251006T140000
DTEND;TZID=Europe/Paris:20251006T140000
DTSTAMP:20260526T142521
CREATED:20250728T121025Z
LAST-MODIFIED:20251003T182358Z
UID:186790-1759759200-1759759200@www.bordeaux-neurocampus.fr
SUMMARY:Thesis defense - Flore Marchaland
DESCRIPTION:Venue: BBS \nDefense in french \n\nFlore Marchaland \nTeam : Brain Nutrient Sensing and Cognitive Trajectory from Development to Aging (NutriMind)\nNutriNeuro \nTitle\nMechanistic role of polyunsaturated fatty acids in brain development and cognitive trajectory \nAbstract\nN-3 and n-6 polyunsaturated fatty acids (PUFA) are essential for mammals\, as they cannot be synthesized de novo and must therefore be provided by the diet. Once consumed\, PUFA are incorporated into cellular membranes\, where they play a major structural role. Among PUFA\, docosahexaenoic acid (DHA\, n-3) and arachidonic acid (ARA\, n-6) are long-chain PUFA (LC-PUFA) that are particularly enriched in the brain. They are respectively derived from the metabolism of α-linolenic acid (ALA\, n-3) and linoleic acid (LA\, n-6)\, their precursors found in plant-derived foods. LC-PUFA can also be directly supplied by animal-derived foods\, primarily marine sources for n-3 and terrestrial sources for n-6. LC-PUFA accumulate in the brain during the perinatal period during which they are transferred from the mother to the infant first via the placenta and subsequently through breast milk. Thus\, the supply of LC-PUFA to the infant largely depends on maternal dietary PUFA intake. However\, since the late 20th century\, changes in dietary habits have led to an increase in n-6 PUFA consumption alongside a decrease in n-3 PUFA intake. Epidemiological and clinical studies have highlighted an inverse correlation between maternal n-3 PUFA dietary intake and children’s cognitive performance. Furthermore\, experimental studies in rodents have shown that perinatal n-3 PUFA deficiency impairs cognitive functions as well as neuronal morphology and function in male offspring. However\, the underlying mechanisms and potential sex-specific differences remain poorly understood. \nIn this context\, the primary objective of my PhD research was to investigate the cellular and molecular mechanisms underlying the behavioral and neurobiological effects induced by perinatal n-3 PUFA deficiency in both male and female offspring. Additionally\, I examined whether maintaining n-3 PUFA through an innovative genetic approach could prevent these alterations. \nThe results obtained indicate that perinatal n-3 PUFA deficiency affects\, in some cases in a sex-dependent manner\, 1) brain profiles of PUFA and their oxygenated derivatives (oxylipins) ; 2) the trajectory of physical and motor development ; 3) spatial memory ; and 4) long-term synaptic plasticity in the hippocampus. Genetic maintenance of PUFA in offspring exposed to this perinatal n-3 PUFA deficiency enables 1) the prevention of impairments in spatial memory and long-term synaptic plasticity in the hippocampus ; 2) an increase in mature synapse density\, particularly glutamatergic synapses\, within the hippocampus ; 3) the promotion of a transcriptional signature associated with synapse and neuronal network maturation ; and 4) the generation of a unique brain oxylipin profile differing by sex. In contrast\, genetic modulation of PUFA synthesis specifically in CaMKII neurons\, which are predominantly glutamatergic\, is not sufficient to prevent alterations in the PUFA profile of hippocampal synaptosomes or to rescue memory deficits induced by perinatal n-3 PUFA deficiency. Finally\, this work also shows that perinatal n-3 PUFA deficiency alters the transcriptomic signature of hippocampal microglia only from three weeks after birth\, with no significant effects observed earlier in development\, in a sex-dependent manner. \nOverall\, my PhD work has shed light on the mechanisms by which perinatal n-3 PUFA deficiency impairs brain development and cognition\, disrupting synaptic and neuronal network maturation in the hippocampus at both the cellular and molecular levels. \nKeywords : Development\, Behavior\, Nutrition\, Microglia\, Synapse \nPublication\nDi Miceli\, M.\, Rossitto\, M.\, Martinat\, M.\, Marchaland\, F.\, Kharbouche\, S.\, Graland\, M.\, … & Layé\, S. (2024). Modified neuroimmune processes and emotional behaviour in weaned and late adolescent male and female mice born via caesarean section. Scientific Reports\, 14(1)\, 29807. \nJury\n\n\n\nDr Sophie LAYÉ\, Directrice de recherche INRAE\, Université de Bordeaux\, France\nDirectrice de thèse\n\n\nDr Pascale CHAVIS\, Directrice de recherche INSERM\, Université Aix-Marseille\, FrancePr Pierre GRESSENS\, Professeur\, Université Paris Diderot\, France \nDr Daniela COTA\, Directrice de recherche INSERM\, Université de Bordeaux\, France\nRapportriceRapporteur \nExaminatrice\n\n\nDr Émilie PACARY\, Chargée de recherche INSERM\, Université de Bordeaux\, France\nExaminatrice\n\n\nPr Christoph RUMMEL\, Professeur\, Université Justus Liebig de Giessen\, AllemagnePr Richard BAZINET\, Professeur\, Université de Toronto\, Canada \nDr Jean-Christophe DELPECH\, Chargé de recherche INRAE\, Université de Bordeaux\, France\nExaminateurInvité \nInvité\n\n\n\n  \n  \n
URL:https://www.bordeaux-neurocampus.fr/en/event/thesis-defense-flore-marchaland/
CATEGORIES:Thesis
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BEGIN:VEVENT
DTSTART;TZID=Europe/Paris:20251006T180000
DTEND;TZID=Europe/Paris:20251006T180000
DTSTAMP:20260526T142521
CREATED:20250909T134329Z
LAST-MODIFIED:20250909T134648Z
UID:187754-1759773600-1759773600@www.bordeaux-neurocampus.fr
SUMMARY:Soirée santé CHU - Mollat : "Quel avenir après un AVC ?"
DESCRIPTION:Lieu : Station Ausone – 8 rue de la Vieille Tour\, Bordeaux \nEgalement sur YouTube \n\nProgramme :\nTémoignage \nMargot Turcat\nà l’occasion de la parution de “Ça me prend la tête” aux éditions Larousse. \nQuel avenir après un AVC ?\nPar Igor Sibon \nLes accidents vasculaires cérébraux affectent près de 150 000 patients en France par an. Les deux dernières décennies ont vu émerger de nouvelles stratégies thérapeutiques telles que la thrombolyse ou la thrombectomie mécanique permettant d’améliorer le pronostic fonctionnel post accident vasculaire cérébral lorsqu’elles sont utilisés dans les premières heures suivant le début des symptômes. Néanmoins\, 2/3 des patients vont potentiellement conserver des séquelles\, qu’elles soient visibles à titre de paralysie ou de troubles du langage ou invisible\, tels que les troubles de la concentration\, de la mémoire ou de l’humeur. Ces différentes séquelles doivent être systématiquement évaluées et prise en charge par des professionnels de santé spécialisés pour optimiser la qualité de vie post accident vasculaire cérébral. \nIgor Sibon est PU PH de Neurologie\, Chef de Service de l’Unité Neurovasculaire du CHU de Bordeaux\, Directeur par Interim de l’IHU VBHI\, Chercheur membre de l’INCIA (coresponsable de l’équipe ECOPSY dont l’objectif est de mieux prédire et prendre en charge les troubles cognitifs et thymiques post-AVC) \nLes séquelles post AVC\, vivre avec même quand elles se cachent\nPar Bertrand Glize \nDans les suites d’un AVC\, et malgré l’amélioration constante de la prise en charge en urgence\, peuvent persister des séquelles\, parfois importantes. Si les troubles moteurs tels qu’une hémiplégie sont largement perçus par tous\, le handicap « invisible » impacte aussi grandement le quotidien des patients. Par exemple\, les troubles du langage tels que l’aphasie (des difficultés à comprendre ou produire les mots et les phrases à l’oral et/ou à l’écrit) sont connus pour impacter la qualité de vie\, parfois plus que des maladies comme le cancer ou la maladie d’Alzheimer selon certaines études. Si la rééducation a pour but de minimiser ces conséquences\, l’accompagnement médical\, mais aussi social et humain est primordial et doit être évoqué librement\, car il reste malgré tout insuffisant. \nBertrand Glize est PU-PH de Médecine Physique et de Réadaptation U1219 INSERM BPH équipe ACTIVE. \n  \n
URL:https://www.bordeaux-neurocampus.fr/en/event/soiree-sante-chu-mollat-quel-avenir-apres-un-avc/
CATEGORIES:Events for all,pour tous homepage
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